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Hydatid Cyst of Liver — A Laparoscopic Management
Amit Goel*, Brijendra Tiwari**, Pavitra Ganguli***
 
Introduction

Hydatid cyst is a parasitic disease caused by Echinococcus granulosus known as classic hydatid disease. Echinococcus multilocularis results in alveolar hydatid disease, a less localised disease and both are continuing public health problem in state of Jammu and Kashmir where sheep herding and breeding is a widespread profession. We present a case report of hydatid disease of liver managed laparoscopically with good results.

 
Case Report
A 32 year male was admitted in our hospital with pain right hypochondrium since 6 months. There was no history of weight loss, fever, urticaria. On examination there was fullness of epigastrium and right hypochondrium. Liver was palpable two fingers at midclavicular line. There was no icterus, rashes and oedema. All routine haematological investigations were normal. Plain film radiography of chest showed a raised right dome of diaphragm. AntiEchinococcus IgG antibodies was 37 u/l [positive 12 u/l]. Ultrasound showed echogenic mass of 9 x 9 cm with multiple daughter cysts and clear fluid.Computed Tomography showed a well defined hypodense lesion of 9 x 9 cm at right lobe of liver with multiple areas of increased densities with no calcifications. Preoperatively patient was put on albendazole 10 mg/kg/day for 10 days. Patient was posted for surgery and laparoscopic cystectomy was planned. Patient was put in supine position with head raised and left lateral tilt. A specially designed port [12 mm] having 15 inches length with a long sheath which reaches into deep seated cavity was used. It has a side channel of same diameter through which a strong suction was done and entire contents of cyst were evacuated. Patient recovered well postoperatively and had no recurrence of symptoms over a follow up of one year.
Fig. 1 : MRI chest and neck (Horizontal Section). Arrow Head : Tumour mass compressing the trachea and oesophagus
 
Discussion

Echinococcus is present in the intestine of carnivore like dog which is the definitive host. Eggs are ingested by herbivore like sheep which is intermediate host and goes to liver through portal vein where it forms hydatid cyst. Man is an accidental host. Most common sites are liver (60%) and lung (30%). Pain in right upper abdomen is the most common symptom. Specificity of ultrasound is 90% and CT scan is 100%. Modes of management have been medical with mebendazole and albendazole. Surgery for hydatid cyst of liver are percutaneous aspiration, marsupilization, partial or total pericystectomy and liver resections. Akin et al concluded in his study that surgical management of hydatid disease without drainage decreases postoperative complication rate and average hospitalization period.1

Demirbilk et al studied and compared the results of medical treatment and various surgical treatment between 1988 and 1997 and concluded that surgical treatment offers early cure than medical treatment.2

Laparoscopic surgery has added new dimension to hydatid cyst surgery. Patients with small size and early stage cysts are ideal cases for laparoscopic treatment as wall collapses after laparoscopic evacuation.

Ertem et al studied 12 patients of hydatid cyst of liver and treated them laparoscopically and concluded that these patients had postoperative comfort, less pain, better scar, decreased hospital stay and early return to daily activities.3

 
Fig. 2 : MRI Chest and Neck (Sagittal Section). Arrow head : Tumour mass in cervical region and superior mediastinum. anterior mediastinum has no thymic tissue
 
Conclusion
Laparoscopic management is an alternative and useful method of treating hydatid cyst of liver. It has results comparable with open surgery and has all benefits of minimal access surgery and is the current treatment of choice for early management of hydatid cyst of liver.
 
References
1. Akin ML, Erenoglu C, Uncu EU, Basekim C, Batkin A. Surgical management of hydatid disease of liver- A military experience. J R Arm Corps 1998; 144 (3) : 139-43.
2. Demirbilek S, Sander S, Atayvrt HF, Ayding G. Hydatid disease of liver in childhood; the success of medical therapy and surgical alternatives. Paediatr Surg Int 2001; 17 (56) : 373-77.
3. Ertem M, Uras C, Karahasanglu T, Erguney S, Alemdaroglu K. Laparoscopic aproach to hepatic hydatid disease. Dig Surg 1998; 15 (4) : 333-36
4. Croese TJ. Eosinophilic enteritis-a recent North Queensland experience. Aust N Z J Med 1998; 18 (7); 848-53.
5. Wing-harkins DL, Dellinger GW, Lynch C, Mihas AA. Eosinophilic gastroenteritis associated with protein losing enteropathy. J Int Med Res 1996; 24 (1) : 155-63.
6. Wig JD, Goenka MK, Bhasin DK, Vajphei K. Eosinophilic gastroenteritis presenting as acute intestinal obstruction. Indian J Gastroenterol 1995; 14 : 104-5.
7. Alexander P, Jacob S, Paul V. Laparoscopy in eosinophilic jejunitis presenting as subacute bowel obstruction: a case report. Tropical Gastroenterology 2003; 24 : 97-98.
8. Kamal MF, Shaker K, Jaser N, Leimoon BA. Eosinophilic gastroenteritis with no peripheral eosinophilia. Ann Chir Gynaecol 1995; 98-100.
9. Klien NC, Hargrove RL, Sleisenger MH. Eosinophilic gastroenteritis. Medicine 1970; 49 : 299-319.
 

Eradicating H. pylori

Does not increase symptoms of gastro-oesophageal reflux disease

Does eradication of Helicobacter pylori lead to an increase in symptoms of gastro-oesophageal reflux disease? We need to know this because the benefits of eradicating H. pylori relative to acid suppression alone are small in non-ulcer dyspepsia and uninvestigated dyspepsia.

Overwhelming evidence now shows that eradicating H. pylori has little or no impact on gastro-oesophageal reflux disease in the general population.

BMJ, 2004; 328 : 1388.