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Airway Management for Laser Excision of Subglottic Stenosis
 
V Chandrashekhar*, M Soumya**, RD Patel***
 

This article sums up the airway management for laser excision of subglottic stenosis following phenol ingestion. Following general anaesthesia there was inability to pass even a 2.5 no. ID red rubber endotracheal tube with metal foil, hence a 3 no. bronchoscope was placed at the glottis and ventilation maintained by Sanders injector with 16G needle. Laser excision of stricture was conducted with the bronchoscope in place and ventilation done intermittently. Perioperatively following widening of the stricture, the bronchoscope was removed and a 6.5 no. ID red rubber endotracheal tube with metal foil was passed and laser excision continued. Post procedure this was replaced with a 7 no. ID portex endotracheal tube. All precautions to provide adequate oxygenation were taken.

 
Introduction

The surgical application of lasers by otolaryngologists is most important to the anaesthesiologist as the airway is shared by both the surgeon and the anaesthesiologist. Laser surgery is best employed to resect subglottic stenosis (Myers and Cotton Grade 2 and 3) and subglottic haemangiomas. The CO2 laser beam is absorbed strongly by water, blood and biological tissue. It produces radiation with a wavelength of 10 mm damaging tissue surfaces to a depth of 200 mm. Hence it is suitable for removing lesions on the vocal cords. Performing CO2 laser airway surgery has a higher rate of ventilatory complications with jet ventilation than with a standard endotracheal tube. For the safe conduct of anaesthesia the technique should be based on the experience of the operator and characteristics of the stenosis. One such management is discussed in the following case report.

 
Case Report

A 20 year old female presented with respiratory distress and stridor following an alleged history of phenol ingestion. An emergency tracheostomy was conducted as intubation was not possible due to distorted airway following corrosive injury due to phenol. Also patient had oesophageal stricture on further investigation following admission. A feeding jejunostomy was done under general anaesthesia. The patient was decannulated of the tracheostomy tube gradually.

But 9 months later patient had episode of respiratory distress but no stridor. So this patient was admitted and an indirect laryngoscopy was done. It revealed that there was very narrow supraglottic opening and sclerotic bands from epiglottis to posterior pharyngeal wall, vallecula and aryepiglottic fold. Hence patient was posted for a laser excision of the obstruction the next day. A high risk consent as well as tracheostomy consent were taken. The patient was monitored with pulse oximetry, non-invasive blood pressure and cardioscope. General anaesthesia was induced with propofol and vecuronium. It was a difficult intubation, hence all sizes of red rubber tubes were kept ready. Mask ventilation was barely possible. Oxygenation was adequately maintained. On direct laryngoscopy the narrow opening did not allow the passage of even the smallest of the red rubber endotracheal tube (2.5 no. ID) with a metal foil wrapped around it probably due to subglottic stenosis which was not anticipated. Since the patient had already undergone a tracheostomy before, there was scarring and hence a higher chance of bleeding and further complication with a repeat tracheostomy. So considering the apnoea time it was decided to place a 3 no. rigid bronchoscope at the opening and try ventilating with a Sanders injector using a 16 no. needle. This technique succeeded and it was decided to proceed with the procedure. The patient’s eyes were taped, closed and covered with moist gauze piece. Nontarget tissue was protected with moist gauze covering. Also alignment of the laser (CO2 laser) was checked and patient immobility ensured (Fig. 1).


Fig. 1 : Patient positioned with bronchoscope, Sander’s jet ventilation and laser apparatus with adequate precautions.

Anaesthesia was maintained with propofol and fentanyl infusion. Laser excisions of the bands were carried out with intermittent ventilation from the side port using the Sanders injector. Once the opening was sufficiently wide (Fig. 2), the bronchoscope was removed and then a 6.5 no. ID red rubber endotracheal tube with a metal foil wrapped around was inserted by direct laryngoscopy and airway was secured. Following complete excision of the stenosis, a 7 no. ID portex endotracheal tube was gently placed after removing the red rubber tube. The patient was reversed and smoothly extubated on table after spraying the cords with lignocaine to avoid post operative croup. Also post operatively humidification of inspired supplemental oxygen was provided.

 
Discussion

A rigid bronchoscope allows better visualization, access, control of bleeding, suctioning and irrigation in laser surgeries.1 Also to reduce the incidence of fire during laser surgery on the airway, not more than 30% oxygen should be used in gas mixture.2 Thus while using the jet venturi ventilation technique the chances of ignition in the airway with the high jet flow of oxygen are variable.3 But the literature on ventilation and anaesthesia technique for bronchoscopic laser surgery is not consistent.

Polyvinyl chloride (PVC) tubes burn more vigorously producing hydrogen chloride which is a pulmonary toxin. Hence it is preferred to intubate patients with red tubber tubes wrapped in metal foil or with silicone tubes.4 Metal tubes are the only non-combustible tubes. But they also have lot of disadvantages.5 In India, we have no experience of metal tubes. Further the management of subglottic laser surgery presents challenges for the anaesthesiologist.


Fig. 2 : View of the subglottic stenosis mid-way through the Laser apparatus showing partial excision of stenosis.

Performing CO2 laser airway surgery has a higher rate of ventilatory complications with jet ventilation than with a standard endotracheal tube. Jet ventilation produces barotrauma to the tracheobronchial tree and lungs if the airway does not remain patent.6 It is necessary that the laryngeal pathology does not interfere with the escape of the jet ventilation gases from the tracheobronchial tree. But mucosal dehydration is common with jet ventilation and leads to increased risk of postoperative laryngeal web formation. Hence good humidification of inspired gases postoperatively and good systemic hydration is desirable. Also particulate blasting of blood and dissected tissue down the tracheal tree can occur with jet ventilation.6-8 It has also been suggested that patients undergoing laser excisions of airway tumours during jet ventilation may absorb carbon monoxide from entrained laser smoke in the pharyngeal area.9 But the biggest advantage is that the patient can be ventilated without an endotracheal tube and without obstructing equipment in the larynx offering unparallel access to laryngotracheobronchial tree for both diagnosis and surgery.10,11

In this case report we have tried to highlight the importance of providing adequate oxygenation and maintaining the airway inspite of being in a state of “can’t intubate and can barely ventilate” initially and still carried out a successful excision of subglottic stenosis by laser surgery without a tracheostomy or any other post operative complications. The risk associated with this type of procedure is higher due to compromised airway and use of laser with jet ventilation as well and hence it is to be undertaken only in the presence of an experienced senior anaesthesiologist.

 
References
1. Rontal E, Rontal M, Wenokur ME, et al. Anesthetic management for tracheobronchial laser surgery. Ann Otol Rhinol Laryngol 1986; 95 : 556.
2. Pashayan AG, Wolf G, Gottschallk A, et al. Anesthetic management guidelines for laser airway surgery : ASTM subcomittee. Anesth Patient Safety Found Newslett, 1993; 8 : 13.
3. Weeks DB. Use of jet Venturi ventilation during microsurgery of the glottis and subglottis. Anesth Rev 1985; 12 : 32.
4. Ossoff RH, Duncavage JA, Eisenman TS, et al. Comparison of tracheal damage from laser-ignited endotracheal tube fires. Ann Otol Rhinol Laryngol 1983; 92 : 333.
5. Fried MP, Mallampati SR, Liu FC, et al. Laser resistant stainless steel endotracheal tube : Experimental and clinical evaluation. Lasers Surg Med 1991; 11 : 301.
6. Cozine K, Stone JG, Shulman S, et al. Ventilatory complications of carbon dioxide laser laryngeal surgery. J Clin Anesth 1991; 3 : 20.
7. Courey MS, Osshoff RH. Laser applications in adult laryngeal surgery. Otolaryngol Clin North Am 1996; 29 : 973.
8. Sosis M. Anesthesia for laser surgery. Anesthesiol Clin North Am 1993; 11 : 578.
9. Goldhill DR, Hill AJ, Whitburn RH, et al. Carboxyhaemoglobin concentrations, pulse oximetry and arterial blood-gas tensions during jet ventilation for laser bronchoscopy. British J Anesthesia 1990; 65 : 749.
10. Bourgain JL, Desruennes E, Fischler M, Ravussin P. Transtracheal high frequency jet ventilation for endoscopic airway surgery : a multicentre study. Br J Anaesth 2001; 87 : 870.
11. Patel A, Randhawa N, Semenov RA. Transtracheal high frequency jet ventilation and iatrogenic injury. Br J Anaesth 2002; 89 : 184.
   

STEROIDS DO NOT PREVENT LONG TERM NERVE DAMAGE IN LEPROSY

Giving prednisolone to patients with leprosy does not prevent long term nerve damage. Smith and colleagues randomised 635 people with newly diagnosed multibacillary leprosy from Bangladesh and Nepal to prophylaxis with low dose prednisolone (20 mg/day for three months) or placebo, in addition to standard multidrug treatment. They found that prednisolone can prevent reaction and impairment of nerve function at four months, but only in people without pre-existing nerve damage, and the effect was not sustained at one year. Routine use of prophylactic steroids in all patients with multibacillary leprosy is not justified, say Lockwood and Kumar in an accompanying editorial.

BMJ, 2004; 328 : 1447,1459.

PANCREATITIS IS MORE COMMON, BUT RESULTS ARE NOT IMPROVING

Pancreatitis has become more common in the past 35 years, but survival has not improved since the 1970s. Goldacre and Roberts found that admissions for acute pancreatitis in southern England increased between 1963 and 1998, from 4.9 to 9.8 per 100 000 people, particularly among the younger age groups. Mortality was 14.2% in 1963-74, 7.6% in 1975-86, and 6.7% in 1987-98. Death rates in the first month after an attack are about 30 times higher than death rates in the general population.

BMJ, 2004; 328 : 1466.


*Lecturer; **Registrar; ***Associate Professor, Department of Anaesthesia,
Seth GS Medical College and KEM Hospital, Parel, Mumbai 400 012.