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| Airway Management for Laser
Excision of Subglottic Stenosis |
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| V Chandrashekhar*, M Soumya**,
RD Patel*** |
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This article sums up the
airway management for laser excision of subglottic stenosis
following phenol ingestion. Following general anaesthesia there
was inability to pass even a 2.5 no. ID red rubber endotracheal
tube with metal foil, hence a 3 no. bronchoscope was placed
at the glottis and ventilation maintained by Sanders injector
with 16G needle. Laser excision of stricture was conducted with
the bronchoscope in place and ventilation done intermittently.
Perioperatively following widening of the stricture, the bronchoscope
was removed and a 6.5 no. ID red rubber endotracheal tube with
metal foil was passed and laser excision continued. Post procedure
this was replaced with a 7 no. ID portex endotracheal tube.
All precautions to provide adequate oxygenation were taken. |
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| Introduction |
The surgical application of lasers by otolaryngologists
is most important to the anaesthesiologist as the airway
is shared by both the surgeon and the anaesthesiologist.
Laser surgery is best employed to resect subglottic stenosis
(Myers and Cotton Grade 2 and 3) and subglottic haemangiomas.
The CO2 laser beam is absorbed strongly by water, blood
and biological tissue. It produces radiation with a wavelength
of 10 mm damaging tissue surfaces to a depth of 200 mm.
Hence it is suitable for removing lesions on the vocal
cords. Performing CO2 laser airway surgery has a higher
rate of ventilatory complications with jet ventilation
than with a standard endotracheal tube. For the safe conduct
of anaesthesia the technique should be based on the experience
of the operator and characteristics of the stenosis. One
such management is discussed in the following case report.
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| Case Report |
A 20 year old female presented with respiratory distress
and stridor following an alleged history of phenol ingestion.
An emergency tracheostomy was conducted as intubation
was not possible due to distorted airway following corrosive
injury due to phenol. Also patient had oesophageal stricture
on further investigation following admission. A feeding
jejunostomy was done under general anaesthesia. The patient
was decannulated of the tracheostomy tube gradually.
But 9 months later patient had episode of respiratory
distress but no stridor. So this patient was admitted
and an indirect laryngoscopy was done. It revealed that
there was very narrow supraglottic opening and sclerotic
bands from epiglottis to posterior pharyngeal wall, vallecula
and aryepiglottic fold. Hence patient was posted for a
laser excision of the obstruction the next day. A high
risk consent as well as tracheostomy consent were taken.
The patient was monitored with pulse oximetry, non-invasive
blood pressure and cardioscope. General anaesthesia was
induced with propofol and vecuronium. It was a difficult
intubation, hence all sizes of red rubber tubes were kept
ready. Mask ventilation was barely possible. Oxygenation
was adequately maintained. On direct laryngoscopy the
narrow opening did not allow the passage of even the smallest
of the red rubber endotracheal tube (2.5 no. ID) with
a metal foil wrapped around it probably due to subglottic
stenosis which was not anticipated. Since the patient
had already undergone a tracheostomy before, there was
scarring and hence a higher chance of bleeding and further
complication with a repeat tracheostomy. So considering
the apnoea time it was decided to place a 3 no. rigid
bronchoscope at the opening and try ventilating with a
Sanders injector using a 16 no. needle. This technique
succeeded and it was decided to proceed with the procedure.
The patient’s eyes were taped, closed and covered
with moist gauze piece. Nontarget tissue was protected
with moist gauze covering. Also alignment of the laser
(CO2 laser) was checked and patient immobility ensured
(Fig. 1).
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Fig. 1 : Patient positioned with bronchoscope, Sander’s jet ventilation and laser apparatus with adequate precautions. |
Anaesthesia was maintained with propofol and fentanyl
infusion. Laser excisions of the bands were carried out
with intermittent ventilation from the side port using
the Sanders injector. Once the opening was sufficiently
wide (Fig. 2), the bronchoscope was removed and then a
6.5 no. ID red rubber endotracheal tube with a metal foil
wrapped around was inserted by direct laryngoscopy and
airway was secured. Following complete excision of the
stenosis, a 7 no. ID portex endotracheal tube was gently
placed after removing the red rubber tube. The patient
was reversed and smoothly extubated on table after spraying
the cords with lignocaine to avoid post operative croup.
Also post operatively humidification of inspired supplemental
oxygen was provided. |
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| Discussion |
A rigid bronchoscope allows better visualization, access,
control of bleeding, suctioning and irrigation in laser
surgeries.1 Also to reduce the incidence of fire during
laser surgery on the airway, not more than 30% oxygen
should be used in gas mixture.2 Thus while using the jet
venturi ventilation technique the chances of ignition
in the airway with the high jet flow of oxygen are variable.3
But the literature on ventilation and anaesthesia technique
for bronchoscopic laser surgery is not consistent.
Polyvinyl chloride (PVC) tubes burn more vigorously producing
hydrogen chloride which is a pulmonary toxin. Hence it
is preferred to intubate patients with red tubber tubes
wrapped in metal foil or with silicone tubes.4 Metal tubes
are the only non-combustible tubes. But they also have
lot of disadvantages.5 In India, we have no experience
of metal tubes. Further the management of subglottic laser
surgery presents challenges for the anaesthesiologist.
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Fig. 2 : View of the subglottic stenosis mid-way through the Laser apparatus showing partial excision of stenosis. |
Performing CO2 laser airway surgery has a higher rate
of ventilatory complications with jet ventilation than
with a standard endotracheal tube. Jet ventilation produces
barotrauma to the tracheobronchial tree and lungs if the
airway does not remain patent.6 It is necessary that the
laryngeal pathology does not interfere with the escape
of the jet ventilation gases from the tracheobronchial
tree. But mucosal dehydration is common with jet ventilation
and leads to increased risk of postoperative laryngeal
web formation. Hence good humidification of inspired gases
postoperatively and good systemic hydration is desirable.
Also particulate blasting of blood and dissected tissue
down the tracheal tree can occur with jet ventilation.6-8
It has also been suggested that patients undergoing laser
excisions of airway tumours during jet ventilation may
absorb carbon monoxide from entrained laser smoke in the
pharyngeal area.9 But the biggest advantage is that the
patient can be ventilated without an endotracheal tube
and without obstructing equipment in the larynx offering
unparallel access to laryngotracheobronchial tree for
both diagnosis and surgery.10,11
In this case report we have tried to highlight the importance
of providing adequate oxygenation and maintaining the
airway inspite of being in a state of “can’t
intubate and can barely ventilate” initially and
still carried out a successful excision of subglottic
stenosis by laser surgery without a tracheostomy or any
other post operative complications. The risk associated
with this type of procedure is higher due to compromised
airway and use of laser with jet ventilation as well and
hence it is to be undertaken only in the presence of an
experienced senior anaesthesiologist. |
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| References |
| 1. |
Rontal E, Rontal M, Wenokur
ME, et al. Anesthetic management for tracheobronchial
laser surgery. Ann Otol Rhinol Laryngol 1986; 95 :
556. |
| 2. |
Pashayan AG, Wolf G, Gottschallk A,
et al. Anesthetic management guidelines for laser
airway surgery : ASTM subcomittee. Anesth Patient
Safety Found Newslett, 1993; 8 : 13. |
| 3. |
Weeks DB. Use of jet Venturi ventilation
during microsurgery of the glottis and subglottis.
Anesth Rev 1985; 12 : 32. |
| 4. |
Ossoff RH, Duncavage JA, Eisenman
TS, et al. Comparison of tracheal damage from laser-ignited
endotracheal tube fires. Ann Otol Rhinol Laryngol
1983; 92 : 333. |
| 5. |
Fried MP, Mallampati SR, Liu FC, et
al. Laser resistant stainless steel endotracheal tube
: Experimental and clinical evaluation. Lasers Surg
Med 1991; 11 : 301. |
| 6. |
Cozine K, Stone JG, Shulman S, et al.
Ventilatory complications of carbon dioxide laser
laryngeal surgery. J Clin Anesth 1991; 3 : 20. |
| 7. |
Courey MS, Osshoff RH. Laser applications
in adult laryngeal surgery. Otolaryngol Clin North
Am 1996; 29 : 973. |
| 8. |
Sosis M. Anesthesia for laser surgery.
Anesthesiol Clin North Am 1993; 11 : 578. |
| 9. |
Goldhill DR, Hill AJ, Whitburn RH,
et al. Carboxyhaemoglobin concentrations, pulse oximetry
and arterial blood-gas tensions during jet ventilation
for laser bronchoscopy. British J Anesthesia 1990;
65 : 749. |
| 10. |
Bourgain JL, Desruennes E, Fischler
M, Ravussin P. Transtracheal high frequency jet ventilation
for endoscopic airway surgery : a multicentre study.
Br J Anaesth 2001; 87 : 870. |
| 11. |
Patel A, Randhawa N, Semenov RA. Transtracheal
high frequency jet ventilation and iatrogenic injury.
Br J Anaesth 2002; 89 : 184. |
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STEROIDS DO NOT PREVENT LONG TERM NERVE DAMAGE IN LEPROSY
Giving prednisolone to patients with leprosy does not prevent long term nerve damage. Smith and colleagues randomised 635 people with newly diagnosed multibacillary leprosy from Bangladesh and Nepal to prophylaxis with low dose prednisolone (20 mg/day for three months) or placebo, in addition to standard multidrug treatment. They found that prednisolone can prevent reaction and impairment of nerve function at four months, but only in people without pre-existing nerve damage, and the effect was not sustained at one year. Routine use of prophylactic steroids in all patients with multibacillary leprosy is not justified, say Lockwood and Kumar in an accompanying editorial.
BMJ, 2004; 328 : 1447,1459.
PANCREATITIS IS MORE COMMON, BUT RESULTS ARE NOT IMPROVING
Pancreatitis has become more common in the past 35 years, but survival has not improved since the 1970s. Goldacre and Roberts found that admissions for acute pancreatitis in southern England increased between 1963 and 1998, from 4.9 to 9.8 per 100 000 people, particularly among the younger age groups. Mortality was 14.2% in 1963-74, 7.6% in 1975-86, and 6.7% in 1987-98. Death rates in the first month after an attack are about 30 times higher than death rates in the general population.
BMJ, 2004; 328 : 1466. |
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*Lecturer; **Registrar; ***Associate Professor, Department of Anaesthesia,
Seth GS Medical College and KEM Hospital, Parel, Mumbai 400 012.
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